RIC3, the cholinergic anti-inflammatory pathway, and neuroinflammation
- 1 June 2020
- journal article
- research article
- Published by Elsevier BV in International Immunopharmacology
- Vol. 83, 106381
- https://doi.org/10.1016/j.intimp.2020.106381
Abstract
No abstract availableKeywords
Funding Information
- United States-Israel Binational Science Foundation (2013055)
- National Institutes of Health (EY024717)
This publication has 37 references indexed in Scilit:
- RIC-3 differentially modulates α4β2 and α7 nicotinic receptor assembly, expression, and nicotine-induced receptor upregulationBMC Neuroscience, 2013
- Inflammatory Mediators Alter the Astrocyte Transcriptome and Calcium Signaling Elicited by Multiple G-Protein-Coupled ReceptorsJournal of Neuroscience, 2012
- Role of HDAC3 on p53 Expression and Apoptosis in T Cells of Patients with Multiple SclerosisPLOS ONE, 2011
- Attenuation of CNS inflammatory responses by nicotine involves α7 and non-α7 nicotinic receptorsExperimental Neurology, 2010
- Ric-3 Promotes α7 Nicotinic Receptor Assembly and Trafficking through the ER Subcompartment of DendritesJournal of Neuroscience, 2010
- Automated High-Throughput siRNA Transfection in Raw 264.7 Macrophages: A Case Study for Optimization ProcedureSLAS Discovery, 2009
- RIC‐3 and nicotinic acetylcholine receptors: Biogenesis, properties, and diversityBiotechnology Journal, 2008
- Ric-3 Promotes Functional Expression of the Nicotinic Acetylcholine Receptor α7 Subunit in Mammalian CellsJournal of Biological Chemistry, 2005
- Conservation within the RIC-3 Gene FamilyJournal of Biological Chemistry, 2003
- Nicotinic acetylcholine receptor α7 subunit is an essential regulator of inflammationNature, 2002