Keap1 governs ageing-induced protein aggregation in endothelial cells
Open Access
- 19 May 2020
- journal article
- research article
- Published by Elsevier BV in Redox Biology
- Vol. 34, 101572
- https://doi.org/10.1016/j.redox.2020.101572
Abstract
No abstract availableKeywords
Funding Information
- National Science Centre (2016/22/E/NZ3/00405)
- National Science Centre (DEC-2019/32/T/NZ3/00326)
- International Associated Laboratory
- CNRS
- European Regional Development Fund and Innovative Economy
- National Cohesion Strategy of Poland
This publication has 53 references indexed in Scilit:
- The Hallmarks of AgingCell, 2013
- Promoter demethylation of Keap1 gene in human diabetic cataractous lensesBiochemical and Biophysical Research Communications, 2012
- Protein misfolding and aggregation in cataract disease and prospects for preventionTrends in Molecular Medicine, 2012
- Aggrephagy: Selective Disposal of Protein Aggregates by MacroautophagyInternational Journal of Cell Biology, 2012
- Keap1 facilitates p62-mediated ubiquitin aggregate clearance via autophagyAutophagy, 2010
- Stress-Activated Cap'n'collar Transcription Factors in Aging and Human DiseaseScience Signaling, 2010
- The selective autophagy substrate p62 activates the stress responsive transcription factor Nrf2 through inactivation of Keap1Nature, 2010
- Mechanisms of Ascorbate-Induced Cytotoxicity in Pancreatic CancerClinical Cancer Research, 2010
- Inflammation and metabolic disordersNature, 2006
- Organism complexity anti‐correlates with proteomic β‐aggregation propensityProtein Science, 2005