Involvement of WAVE Accumulation in Aβ/APP Pathology-Dependent Tangle Modification in Alzheimer’s Disease
- 31 July 2009
- journal article
- research article
- Published by Elsevier BV in The American Journal of Pathology
- Vol. 175 (1), 17-24
- https://doi.org/10.2353/ajpath.2009.080908
Abstract
No abstract availableKeywords
This publication has 31 references indexed in Scilit:
- Co-Localization of Amyloid Beta and Tau Pathology in Alzheimer's Disease SynaptosomesThe American Journal of Pathology, 2008
- p21-activated Kinase-aberrant Activation and Translocation in Alzheimer Disease PathogenesisJournal of Biological Chemistry, 2008
- Females exhibit more extensive amyloid, but not tau, pathology in an Alzheimer transgenic modelBrain Research, 2008
- Collapsin response mediator protein‐2 hyperphosphorylation is an early event in Alzheimer’s disease progressionJournal of Neurochemistry, 2007
- Activation of GSK-3 and phosphorylation of CRMP2 in transgenic mice expressing APP intracellular domainThe Journal of cell biology, 2005
- Rapid WAVE dynamics in dendritic spines of cultured hippocampal neurons is mediated by actin polymerizationJournal of Neurochemistry, 2005
- CRMP-2 binds to tubulin heterodimers to promote microtubule assemblyNature, 2002
- Neuronal loss correlates with but exceeds neurofibrillary tangles in Alzheimer's diseaseAnnals of Neurology, 1997
- τ Protein Kinase II Is Involved in the Regulation of the Normal Phosphorylation State of τ ProteinJournal of Neurochemistry, 1993
- Microtubular reorganization and dendritic growth response in alzheimer's diseaseAnnals of Neurology, 1989