Lithium-induced activation of Akt and CaM kinase II contributes to its neuroprotective action in a rat microsphere embolism model
- 1 September 2006
- journal article
- Published by Elsevier BV in Brain Research
- Vol. 1108 (1), 98-106
- https://doi.org/10.1016/j.brainres.2006.06.009
Abstract
No abstract availableKeywords
This publication has 46 references indexed in Scilit:
- Glycogen synthase kinase 3: a drug target for CNS therapiesJournal of Neurochemistry, 2004
- 14-3-3 Connects Glycogen Synthase Kinase-3β to Tau within a Brain Microtubule-associated Tau Phosphorylation ComplexJournal of Biological Chemistry, 2003
- Cell cycle and death control: long live ForkheadsTrends in Biochemical Sciences, 2002
- Neuroprotective effects of lithium in cultured cells and animal models of diseasesBipolar Disorders, 2002
- Role of protein phosphatase‐2A and ‐1 in the regulation of GSK‐3, cdk5 and cdc2 and the phosphorylation of tau in rat forebrainFEBS Letters, 2000
- Regulation and localization of tyrosine216phosphorylation of glycogen synthase kinase-3β in cellular and animal models of neuronal degenerationProceedings of the National Academy of Sciences, 2000
- Lithium activates the serine/threonine kinase Akt-1 and suppresses glutamate-induced inhibition of Akt-1 activity in neuronsProceedings of the National Academy of Sciences, 1999
- Regulation of Cell Death Protease Caspase-9 by PhosphorylationScience, 1998
- Regulated phosphorylation and dephosphorylation of tau protein: effects on microtubule interaction, intracellular trafficking and neurodegenerationBiochemical Journal, 1997
- Fodrin degradation and subcellular distribution of calpains after neonatal rat cerebral hypoxic-ischemiaBrain Research, 1995