Leukemia translocation protein PLZF inhibits cell growth and expression of cyclin A
- 28 January 1999
- journal article
- Published by Springer Science and Business Media LLC in Oncogene
- Vol. 18 (4), 925-934
- https://doi.org/10.1038/sj.onc.1202375
Abstract
Oncogene is one of the world’s leading cancer journals. It is published weekly and covers all aspects of the structure and function of Oncogenes. Oncogene also publishes 8 Reviews issues a year, on a broad range of topics.Keywords
This publication has 92 references indexed in Scilit:
- Phylogenomics reveals a new ‘megagroup’ including most photosynthetic eukaryotesBiology Letters, 2008
- Differential Requirements for MCM Proteins in DNA Replication in Drosophila S2 CellsPLOS ONE, 2007
- Toward Resolving the Eukaryotic Tree: The Phylogenetic Positions of Jakobids and CercozoansCurrent Biology, 2007
- Genome-Wide Analysis of the Core DNA Replication Machinery in the Higher Plants Arabidopsis and RicePlant Physiology, 2007
- Identification of a novel cell-cycle-induced MCM family protein MCM9Biochemical and Biophysical Research Communications, 2005
- MUSCLE: multiple sequence alignment with high accuracy and high throughputNucleic Acids Research, 2004
- Fusion of retinoic acid receptor α to NuMA, the nuclear mitotic apparatus protein, by a variant translocation in acute promyelocytic leukaemiaNature Genetics, 1997
- Cancer Cell CyclesScience, 1996
- PLZF-RAR alpha fusion proteins generated from the variant t(11;17)(q23;q21) translocation in acute promyelocytic leukemia inhibit ligand-dependent transactivation of wild-type retinoic acid receptors.Proceedings of the National Academy of Sciences of the United States of America, 1994
- Cyclin A is required in S phase in normal epithelial cellsBiochemical and Biophysical Research Communications, 1992