The Nonreceptor Protein-tyrosine Kinase c-Fes Is Involved in Fibroblast Growth Factor-2-induced Chemotaxis of Murine Brain Capillary Endothelial Cells
Open Access
- 1 April 2000
- journal article
- Published by Elsevier BV
- Vol. 275 (14), 10105-10111
- https://doi.org/10.1074/jbc.275.14.10105
Abstract
No abstract availableKeywords
This publication has 28 references indexed in Scilit:
- Fibroblast Growth Factor Receptor-1-mediated Endothelial Cell Proliferation Is Dependent on the Src Homology (SH) 2/SH3 Domain-containing Adaptor Protein CrkJournal of Biological Chemistry, 1999
- Contribution of Src and Ras pathways in FGF-2 induced endothelial cell differentiationOncogene, 1999
- Fibroblast growth factor receptor-1 mediates chemotaxis independently of direct SH2-domain protein bindingOncogene, 1998
- Activation of STAT3 by the c-Fes Protein-tyrosine KinaseJournal of Biological Chemistry, 1998
- Autophosphorylation of the Fes Tyrosine KinasePublished by Elsevier BV ,1996
- Shc and a Novel 89-kDa Component Couple to the Grb2-Sos Complex in Fibroblast Growth Factor-2-stimulated CellsPublished by Elsevier BV ,1995
- Endothelial receptor tyrosine kinases involved in angiogenesis.The Journal of cell biology, 1995
- Inhibition of c-fes expression by an antisense oligomer causes apoptosis of HL60 cells induced to granulocytic differentiation.The Journal of Experimental Medicine, 1993
- Diverse receptors for fibroblast growth factorsProgress in Growth Factor Research, 1992
- Point mutation of an FGF receptor abolishes phosphatidylinositol turnover and Ca2+ flux but not mitogenesisNature, 1992