Increase in brain 125I-cholecystokinin (CCK) receptor binding following chronic haloperidol treatment, intracisternal 6-hydroxydopamine or ventral tegmental lesions
- 1 February 1983
- journal article
- research article
- Published by Elsevier BV in Life Sciences
- Vol. 32 (8), 871-878
- https://doi.org/10.1016/0024-3205(83)90224-2
Abstract
No abstract availableKeywords
This publication has 18 references indexed in Scilit:
- Peptide-monoamine coexistence: Studies of the actions of cholecystokinin-like peptide on the electrical activity of midbrain dopamine neuronsNeuroscience, 1981
- Changes in brain cholecystokinin octapeptide following lesions of the medial forebrain bundleBrain Research, 1981
- Effects of cholecystokinin octapeptide on striatal dopamine metabolism and on apomorphine-induced stereotyped cage-climbing in miceEuropean Journal of Pharmacology, 1981
- A subpopulation of mesencephalic dopamine neurons projecting to limbic areas contains a cholecystokinin-like peptide: Evidence from immunohistochemistry combined with retrograde tracingNeuroscience, 1980
- Cholecystokinin peptides produce marked reduction of dopamine turnover in discrete areas in the rat brain following intraventricular injectionEuropean Journal of Pharmacology, 1980
- Evidence for coexistence of dopamine and CCK in meso-limbic neuronesNature, 1980
- Brain neurotransmitter receptors after long-term haloperidol: Dopamine, acetylcholine, serotonin, α-noradrenergic and naloxone receptorsLife Sciences, 1977
- Antischizophrenic Drugs: Chronic Treatment Elevates Dopamine Receptor Binding in BrainScience, 1977
- Stereotaxic Mapping of the Monoamine Pathways in the Rat Brain*Acta Physiologica Scandinavica, 1971
- 6-hydroxy-dopamine induced degeneration of central monoamine neuronsEuropean Journal of Pharmacology, 1968