Astrocytosis, βA4-protein deposition and paired helical filament formation in Alzheimer's disease
- 31 October 1992
- journal article
- Published by Elsevier BV in Journal of the Neurological Sciences
- Vol. 112 (1-2), 68-75
- https://doi.org/10.1016/0022-510x(92)90134-7
Abstract
No abstract availableKeywords
This publication has 38 references indexed in Scilit:
- Segregation of a missense mutation in the amyloid precursor protein gene with familial Alzheimer's diseaseNature, 1991
- Large amounts of neocortical βA4 deposits without neuritic plaques nor tangles in a psychometrically assessed, non-demented personNeuroscience Letters, 1990
- Expression of β-amyloid precursor protein in reactive astrocytes following neuronal damageNeuron, 1989
- Neurotoxicity of a Fragment of the Amyloid Precursor Associated with Alzheimer's DiseaseScience, 1989
- Recommended minimum data to be collected in research studies on Alzheimer's disease. The MRC (UK) Alzheimer's Disease Workshop Steering Committee.Journal of Neurology, Neurosurgery & Psychiatry, 1989
- Patterns of gliosis in alzheimer's disease and aging cerebrumGlia, 1989
- Amyloid β Protein Gene: cDNA, mRNA Distribution, and Genetic Linkage Near the Alzheimer LocusScience, 1987
- A MONOCLONAL ANTIBODY TO AMYLOID IN THE BRAINS OF PATIENTS WITH ALZHEIMER'S DISEASENeuropathology and Applied Neurobiology, 1986
- Occurrence of neuropil threads in the senile human brain and in Alzheimer's disease: A third location of paired helical filaments outside of neurofibrillary tangles and neuritic plaquesNeuroscience Letters, 1986
- ALZHEIMER’S DISEASE —AN ELECTRON MICROSCOPICAL STUDYBrain, 1964