Reactive oxygen species mediated NF-κB/p38 feedback loop implicated in proliferation inhibition of HFLS-RA cells induced by 1,7-dihydroxy-3,4-dimethoxyxanthone
- 1 October 2017
- journal article
- Published by Elsevier BV in Biomedicine & Pharmacotherapy
- Vol. 94, 1002-1009
- https://doi.org/10.1016/j.biopha.2017.07.164
Abstract
No abstract availableKeywords
Funding Information
- State Oceanic Administration (2012-32)
- State Administration of Traditional Chinese Medicine of the People's Republic of China
- National Natural Science Foundation of China (81603388)
- Natural Science Foundation of Anhui Province (1508085SQH226)
This publication has 26 references indexed in Scilit:
- Advanced Glycation End Products Induce Human Corneal Epithelial Cells Apoptosis through Generation of Reactive Oxygen Species and Activation of JNK and p38 MAPK PathwaysPLOS ONE, 2013
- The p38 MAPK Pathway in Rheumatoid Arthritis: A Sideways LookThe Open Rheumatology Journal, 2012
- Effects of gambogic acid on the activation of caspase-3 and downregulation of SIRT1 in RPMI-8226 multiple myeloma cells via the accumulation of ROSOncology Letters, 2012
- Cluvenone induces apoptosis via a direct target in mitochondria: a possible mechanism to circumvent chemo-resistance?Investigational New Drugs, 2011
- Constitutive intestinal NF-κB does not trigger destructive inflammation unless accompanied by MAPK activationThe Journal of Experimental Medicine, 2011
- Recent cancer drug development with xanthone structuresJournal of Pharmacy and Pharmacology, 2009
- Developments in the clinical understanding of rheumatoid arthritisArthritis Research & Therapy, 2009
- NF-κB as a potential therapeutic target in osteoarthritis and rheumatoid arthritisOsteoarthritis and Cartilage, 2006
- ERK1/2 and p38-MAPK signalling pathways, through MSK1, are involved in NF-κB transactivation during oxidative stress in skeletal myoblastsCellular Signalling, 2006
- Baseline comorbidity levels in biologic and standard DMARD treated patients with rheumatoid arthritis: results from a national patient registerAnnals Of The Rheumatic Diseases, 2005