Mismatch repair deficiency is an uncommon mechanism of alkylator resistance in pediatric malignant gliomas: A report from the children's oncology group
Open Access
- 29 June 2010
- journal article
- research article
- Published by Wiley in Pediatric Blood & Cancer
- Vol. 55 (6), 1066-1071
- https://doi.org/10.1002/pbc.22634
Abstract
No abstract availableKeywords
This publication has 94 references indexed in Scilit:
- Toggling between gamma-frequency activity and suppression of cell assembliesFrontiers in Computational Neuroscience, 2013
- Minimal Size of Cell Assemblies Coordinated by Gamma OscillationsPLoS Computational Biology, 2012
- Synchronization of Firing in Cortical Fast-Spiking Interneurons at Gamma Frequencies: A Phase-Resetting AnalysisPLoS Computational Biology, 2010
- MSH6 Mutations Arise in Glioblastomas during Temozolomide Therapy and Mediate Temozolomide ResistanceClinical Cancer Research, 2009
- Phase-Resetting Curves Determine Synchronization, Phase Locking, and Clustering in Networks of Neural OscillatorsJournal of Neuroscience, 2009
- Gamma oscillations mediate stimulus competition and attentional selection in a cortical network modelProceedings of the National Academy of Sciences of the United States of America, 2008
- An Integrated Genomic Analysis of Human Glioblastoma MultiformeScience, 2008
- Mismatch Repair Deficiency Does Not Mediate Clinical Resistance to Temozolomide in Malignant GliomaClinical Cancer Research, 2008
- Radiotherapy plus Concomitant and Adjuvant Temozolomide for GlioblastomaNew England Journal of Medicine, 2005
- Effects of Noisy Drive on Rhythms in Networks of Excitatory and Inhibitory NeuronsNeural Computation, 2005