ERK and PI3K regulate different aspects of the epithelial to mesenchymal transition of mammary tumor cells induced by truncated MUC1
- 1 May 2009
- journal article
- Published by Elsevier BV in Experimental Cell Research
- Vol. 315 (8), 1490-1504
- https://doi.org/10.1016/j.yexcr.2009.02.011
Abstract
No abstract availableKeywords
This publication has 57 references indexed in Scilit:
- Overexpression and altered glycosylation of MUC1 in malignant mesotheliomaBritish Journal of Cancer, 2008
- Epithelial-Mesenchymal Transition in Breast Cancer Relates to the Basal-like PhenotypeCancer Research, 2008
- Cellular and molecular mechanisms of fibrosisThe Journal of Pathology, 2007
- Snail is required for transforming growth factor-β-induced epithelial–mesenchymal transition by activating PI3 kinase/Akt signal pathwayBiochemical and Biophysical Research Communications, 2006
- ERK1 and ERK2 mitogen-activated protein kinases affect Ras-dependent cell signaling differentiallyJournal of Biology, 2006
- Autoproteolysis coupled to protein folding in the SEA domain of the membrane-bound MUC1 mucinNature Structural & Molecular Biology, 2005
- The MAP kinase pathway is required for entry into mitosis and cell survivalOncogene, 2004
- CA27.29: a valuable marker for breast cancer management. A confirmatory multicentric study on 603 casesEuropean Journal Of Cancer, 2001
- The transcription factor Snail is a repressor of E-cadherin gene expression in epithelial tumour cellsNature, 2000
- Detection of a Secreted MUC1/SEC Protein by MUC1 Isoform Specific Monoclonal AntibodiesBiochemical and Biophysical Research Communications, 1996