Cytokine-Like Factor 1 Gene Expression Is Enriched in Idiopathic Pulmonary Fibrosis and Drives the Accumulation of CD4+ T Cells in Murine Lungs
Open Access
- 19 March 2012
- journal article
- Published by Elsevier BV in The American Journal of Pathology
- Vol. 180 (5), 1963-1978
- https://doi.org/10.1016/j.ajpath.2012.01.010
Abstract
No abstract availableThis publication has 82 references indexed in Scilit:
- Evolving Genomic Approaches to Idiopathic Pulmonary Fibrosis: Moving Beyond GenesClinical and Translational Science, 2011
- Cytokine-like factor 1 (CLF1): Life after development?Cytokine, 2011
- Differential secretion of the mutated protein is a major component affecting phenotypic severity in CRLF1-associated disordersEuropean Journal of Human Genetics, 2011
- Inhibition and Role of let-7d in Idiopathic Pulmonary FibrosisAmerican Journal of Respiratory and Critical Care Medicine, 2010
- Repression of IP-10 by Interactions between Histone Deacetylation and Hypermethylation in Idiopathic Pulmonary FibrosisMolecular and Cellular Biology, 2010
- Bleomycin and IL-1β–mediated pulmonary fibrosis is IL-17A dependentThe Journal of Experimental Medicine, 2010
- Systematic and integrative analysis of large gene lists using DAVID bioinformatics resourcesNature Protocols, 2008
- Alveolar Macrophages Are a Major Determinant of Early Responses to Viral Lung Infection but Do Not Influence Subsequent Disease DevelopmentJournal of Virology, 2008
- Mutations in Cytokine Receptor-Like Factor 1 (CRLF1) Account for Both Crisponi and Cold-Induced Sweating SyndromesAmerican Journal of Human Genetics, 2007
- Adenosine regulation of alveolar fluid clearanceProceedings of the National Academy of Sciences, 2007