NEURONAL‐GLIAL CONTRIBUTIONS TO TRANSMITTER AMINO ACID METABOLISM: STUDIES WITH KAINIC ACID‐INDUCED LESIONS OF RAT STRIATUM
- 5 October 1979
- journal article
- research article
- Published by Wiley in Journal of Neurochemistry
- Vol. 33 (4), 839-844
- https://doi.org/10.1111/j.1471-4159.1979.tb09913.x
Abstract
No abstract availableThis publication has 28 references indexed in Scilit:
- THE SYNTHESIS OF GLUTAMATE BY RAT BRAIN MITOCHONDRIAJournal of Neurochemistry, 1978
- GLUTAMINE—A MAJOR SUBSTRATE FOR NERVE ENDINGSJournal of Neurochemistry, 1978
- AMINO ACID METABOLISM IN GLIAL CELLS: HOMEOSTATIC REGULATION OF INTRA‐ and EXTRACELLULAR MILIEU BY C‐6 GLIOMA CELLSJournal of Neurochemistry, 1978
- Clinical, neuropathologic and pharmacologic aspects of Huntington's disease: Correlates with a new animal modelProgress in Neuro-Psychopharmacology, 1977
- Duplication of biochemical changes of Huntington's chorea by intrastriatal injections of glutamic and kainic acidsNature, 1976
- Lesion of striatal neurons with kainic acid provides a model for Huntington's choreaNature, 1976
- On glutaminase activity in mammalian synaptosomesBrain Research, 1976
- FATE OF l‐GLUTAMATE IN THE BRAINJournal of Neurochemistry, 1974
- ACTIVATORS and INHIBITORS OF BRAIN GLUTAMINASEJournal of Neurochemistry, 1969
- COMPARTMENTATION OF GLUTAMIC ACID METABOLISM IN BRAIN SLICES*Journal of Neurochemistry, 1968