Clonal analyses define the relationships between chromosomal abnormalities and JAK2V617F in patients with Ph-negative myeloproliferative neoplasms
- 31 October 2009
- journal article
- Published by Elsevier BV in Experimental Hematology
- Vol. 37 (10), 1194-1200
- https://doi.org/10.1016/j.exphem.2009.07.003
Abstract
No abstract availableKeywords
This publication has 21 references indexed in Scilit:
- Clonal analysis of deletions on chromosome 20q and JAK2-V617F in MPD suggests that del20q acts independently and is not one of the predisposing mutations for JAK2-V617FBlood, 2009
- Clonal diversity in the myeloproliferative neoplasms: independent origins of genetically distinct clonesBritish Journal of Haematology, 2009
- Numerical gain and structural rearrangements of JAK2, identified by FISH, characterize both JAK2617V>F-positive and -negative patients with Ph-negative MPD, myelodysplasia, and B-lymphoid neoplasmsExperimental Hematology, 2007
- Role of JAK2 in the pathogenesis and therapy of myeloproliferative disordersNature Reviews Cancer, 2007
- Polycythemia vera is not initiated by JAK2 mutationExperimental Hematology, 2007
- The Myeloproliferative DisordersNew England Journal of Medicine, 2006
- Involvement of various hematopoietic-cell lineages by the JAK2V617F mutation in polycythemia veraBlood, 2006
- Cytogenetic and Molecular Genetic Abnormalities in Agnogenic Myeloid MetaplasiaSeminars in Oncology, 2005
- A Gain-of-Function Mutation ofJAK2in Myeloproliferative DisordersNew England Journal of Medicine, 2005
- A unique clonal JAK2 mutation leading to constitutive signalling causes polycythaemia veraNature, 2005