Aberrant Expression of the Cell Cycle Associated Proteins TP53, MDM2, p21, p27, cdk4, Cyclin D1, RB, and EGFR in Cervical Carcinomas
- 31 May 1999
- journal article
- Published by Elsevier BV in Gynecologic Oncology
- Vol. 73 (2), 223-228
- https://doi.org/10.1006/gyno.1999.5346
Abstract
No abstract availableKeywords
This publication has 20 references indexed in Scilit:
- Interaction between the retinoblastoma protein and the oncoprotein MDM2Nature, 1995
- The retinoblastoma protein and cell cycle controlCell, 1995
- A novel inhibitor of cyclin-Cdk activity detected in transforming growth factor beta-arrested epithelial cells.Molecular and Cellular Biology, 1994
- p53 and E2F-1 cooperate to mediate apoptosis.Proceedings of the National Academy of Sciences, 1994
- p27Kip1, a cyclin-Cdk inhibitor, links transforming growth factor-beta and contact inhibition to cell cycle arrest.Genes & Development, 1994
- The p21 Cdk-interacting protein Cip1 is a potent inhibitor of G1 cyclin-dependent kinasesCell, 1993
- Genetic alterations of the TP53 gene, p53 protein expression and hpv infection in primary cervical carcinomasThe Journal of Pathology, 1993
- Phosphorylation of the retinoblastoma protein by cdk2.Proceedings of the National Academy of Sciences, 1992
- Amplification of a gene encoding a p53-associated protein in human sarcomasNature, 1992
- The mdm-2 oncogene product forms a complex with the p53 protein and inhibits p53-mediated transactivationCell, 1992