Homolateral ataxia and crural paresis is a recognized vascular syndrome. However, confirmation of the causative lesion rests principally on one earlier case with multiple other infarcts. We studied a patient with the clinical syndrome; computerized tomography revealed a lucency that appeared within 1 week of the infarct. Localization of the lesion to the superior portion of the posterior limb of the internal capsule and thalamus is in accord with the original conclusions of Fisher and Cole.1