FGF19-induced Hepatocyte Proliferation Is Mediated through FGFR4 Activation
Open Access
- 1 February 2010
- journal article
- Published by Elsevier BV in Journal of Biological Chemistry
- Vol. 285 (8), 5165-5170
- https://doi.org/10.1074/jbc.m109.068783
Abstract
No abstract availableKeywords
This publication has 19 references indexed in Scilit:
- Selective activation of FGFR4 by an FGF19 variant does not improve glucose metabolism in ob/ob miceProceedings of the National Academy of Sciences of the United States of America, 2009
- Fibroblast Growth Factor 21 Reverses Hepatic Steatosis, Increases Energy Expenditure, and Improves Insulin Sensitivity in Diet-Induced Obese MiceDiabetes, 2009
- C-terminal Tail of FGF19 Determines Its Specificity toward Klotho Co-receptorsPublished by Elsevier BV ,2008
- Resident hepatocyte fibroblast growth factor receptor 4 limits hepatocarcinogenesisMolecular Carcinogenesis, 2008
- Liver-specific Activities of FGF19 Require Klotho betaPublished by Elsevier BV ,2007
- Tissue-specific Expression of βKlotho and Fibroblast Growth Factor (FGF) Receptor Isoforms Determines Metabolic Activity of FGF19 and FGF21Journal of Biological Chemistry, 2007
- βKlotho is required for metabolic activity of fibroblast growth factor 21Proceedings of the National Academy of Sciences, 2007
- Fibroblast growth factor 15 functions as an enterohepatic signal to regulate bile acid homeostasisCell Metabolism, 2005
- FGF-21 as a novel metabolic regulatorJCI Insight, 2005
- A Mouse Model of Hepatocellular CarcinomaThe American Journal of Pathology, 2002