Sphingomyelinase Activity Is Enhanced in Cerebral Cortex of Senescence-Accelerated Mouse-P/10 with Advancing Age
- 28 August 1997
- journal article
- Published by Elsevier BV in Biochemical and Biophysical Research Communications
- Vol. 237 (3), 583-587
- https://doi.org/10.1006/bbrc.1997.7133
Abstract
No abstract availableKeywords
Funding Information
- Ministry of Education
- Korea Science and Engineering Foundation (1993-1996, 961-0719-118-2)
This publication has 20 references indexed in Scilit:
- Senescence-accelerated mouse (SAM): A novel murine model of senescenceExperimental Gerontology, 1997
- Sphingomyelin breakdown and cell fateTrends in Biochemical Sciences, 1996
- Acid Sphingomyelinase–Deficient Human Lymphoblasts and Mice Are Defective in Radiation-Induced ApoptosisCell, 1996
- Ceramide signalling and the immune responseBiochimica et Biophysica Acta (BBA) - Lipids and Lipid Metabolism, 1996
- Role of Ceramide in Cellular SenescenceJournal of Biological Chemistry, 1995
- Ceramide: an intracellular signal for apoptosisTrends in Biochemical Sciences, 1995
- Oxidants, antioxidants, and the degenerative diseases of aging.Proceedings of the National Academy of Sciences, 1993
- Inbred SAM-P/10 as a Mouse Model of Spontaneous, Inherited Brain AtrophyJournal of Neuropathology and Experimental Neurology, 1992
- DNA damage metabolism and agingMutation Research/DNAging, 1990
- The Metabolism of SphingomyelinPublished by Elsevier BV ,1966