Accumulation of Cytotoxins During the Development of Seizures and Edema after Hypoxic-Ischemic Injury in Late Gestation Fetal Sheep
- 1 May 1996
- journal article
- Published by Springer Science and Business Media LLC in Pediatric Research
- Vol. 39 (5), 791-797
- https://doi.org/10.1203/00006450-199605000-00008
Abstract
No abstract availableKeywords
This publication has 37 references indexed in Scilit:
- Mechanisms of delayed cell death following hypoxic-ischemic injury in the immature rat: evidence for apoptosis during selective neuronal lossMolecular Brain Research, 1995
- Delayed (“Secondary”) Cerebral Energy Failure after Acute Hypoxia-Ischemia in the Newborn Piglet: Continuous 48-Hour Studies by Phosphorus Magnetic Resonance SpectroscopyPediatric Research, 1994
- When and Why Do Brain Cells Die?Developmental Medicine and Child Neurology, 1992
- Suppression of postischemic epileptiform activity with MK‐801 improves neural outcome in fetal sheepAnnals of Neurology, 1992
- Microglial-produced nitric oxide and reactive nitrogen oxides mediate neuronal cell deathBrain Research, 1992
- Peroxynitrite-induced membrane lipid peroxidation: The cytotoxic potential of superoxide and nitric oxideArchives of Biochemistry and Biophysics, 1991
- Time course of intracellular edema and epileptiform activity following prenatal cerebral ischemia in sheep.Stroke, 1991
- Delayed Seizures Occurring with Hypoxic- Ischemic Encephalopathy in the Fetal SheepPediatric Research, 1990
- Ionic Composition of Microdialysis Perfusing Solution Alters the Pharmacological Responsiveness and Basal Outflow of Striatal DopamineJournal of Neurochemistry, 1989
- Prognosis of Newborn Infants with Hypoxic-Ischemic Brain Injury Assessed by Phosphorus Magnetic Resonance SpectroscopyPediatric Research, 1989