Cytokine-induced nitric oxide synthase gene transcription is blocked by the heat shock response in human liver cells
- 31 August 1996
- journal article
- Published by Elsevier BV in Surgery
- Vol. 120 (2), 144-149
- https://doi.org/10.1016/s0039-6060(96)80281-9
Abstract
No abstract availableKeywords
This publication has 8 references indexed in Scilit:
- Heat shock protein 70 overexpression affects the response to ultraviolet light in murine fibroblasts. Evidence for increased cell viability and suppression of cytokine release.JCI Insight, 1995
- Endotoxin-stimulated nitric oxide production increases injury and reduces rat liver chemiluminescence during reperfusionGastroenterology, 1995
- Clinical implications of the stress response.JCI Insight, 1995
- Differential Induction of Nitric Oxide Synthase in Hepatocytes During Endotoxemia and the Acute-Phase ResponseArchives of Surgery, 1994
- Independent regulation of prostaglandin production and the stress response in human fibroblastsJournal of Cellular Physiology, 1992
- Stimulation of the nitric oxide synthase pathway in human hepatocytes by cytokines and endotoxin.The Journal of Experimental Medicine, 1992
- Transcriptional inhibition of endotoxin–induced monokine synthesis following heat shock in murine peritoneal macrophagesJournal of Leukocyte Biology, 1992
- Reversal of endotoxin-mediated shock by NG-methyl-L-arginine, an inhibitor of nitric oxide synthesisBiochemical and Biophysical Research Communications, 1990