T cell activation defect in hemodialysis patients: Evidence for a role of the B7/CD28 pathway
- 1 August 1993
- journal article
- Published by Elsevier BV in Kidney International
- Vol. 44 (2), 359-365
- https://doi.org/10.1038/ki.1993.252
Abstract
No abstract availableKeywords
This publication has 19 references indexed in Scilit:
- Functional characterization of a novel anti‐B7 monoclonal antibodyEuropean Journal of Immunology, 1992
- Coexpression and functional cooperation of CTLA-4 and CD28 on activated T lymphocytes.The Journal of Experimental Medicine, 1992
- CTLA-4 is a second receptor for the B cell activation antigen B7.The Journal of Experimental Medicine, 1991
- Regulation of interleukin-2 and interferon-γ gene expression in renal failureKidney International, 1991
- Immune Deficiency in Uremia: Interleukin-2 Production and Responsiveness and Interleukin-2 Receptor Expression and ReleaseNephron, 1991
- Hepatitis B vaccination and interleukin 2 receptor expression in chronic renal failureKidney International, 1990
- Altered monocyte function in uremiaClinical Immunology and Immunopathology, 1990
- A Cell Culture Model for T Lymphocyte Clonal AnergyScience, 1990
- In vivo T cell preactivation in chronic uremic hemodialyzed and non-hemodialyzed patientsKidney International, 1989
- Selective blockade of the antigen-receptor-mediated pathway of T cell activation in patients with impaired primary immune responses.JCI Insight, 1987