Tumor Necrosis Factor-α Signaling via TNFR1/p55 Is Deleterious Whereas TNFR2/p75 Signaling Is Protective in Adult Infarct Myocardium
- 22 October 2010
- book chapter
- conference paper
- Published by Springer Science and Business Media LLC in Advances in experimental medicine and biology
- Vol. 691, 433-448
- https://doi.org/10.1007/978-1-4419-6612-4_45
Abstract
No abstract availableKeywords
This publication has 52 references indexed in Scilit:
- The role of TNF-α receptors p55 and p75 in acute myocardial ischemia/reperfusion injury and late preconditioningJournal of Molecular and Cellular Cardiology, 2008
- TNFR gene-modified mesenchymal stem cells attenuate inflammation and cardiac dysfunction following MIScandinavian Cardiovascular Journal, 2008
- Tumor Necrosis Factor-α Receptor p75 Is Required in Ischemia-Induced NeovascularizationCirculation, 2007
- Aging and angiogenesisFrontiers in Bioscience-Landmark, 2003
- Aging and Survival of Cutaneous MicrovasculatureJournal of Investigative Dermatology, 2002
- Tumor necrosis factor-α-induced apoptosis in T cells from aged humans: a role of TNFR-I and downstream signaling moleculesExperimental Gerontology, 2001
- Therapeutic angiogenesis. A single intraarterial bolus of vascular endothelial growth factor augments revascularization in a rabbit ischemic hind limb model.JCI Insight, 1994
- Age-related impairment of early and late events of signal transduction in mouse immune cellsLife Sciences, 1993
- Hypertension-induced changes of platelet-derived growth factor receptor expression in rat aorta and heart.Hypertension, 1991
- Decrease in capillary growth during agingExperimental Gerontology, 1980