Hypothermic circulatory arrest causes multisystem vascular endothelial dysfunction and apoptosis
- 1 March 2000
- journal article
- Published by Elsevier in The Annals of Thoracic Surgery
- Vol. 69 (3), 696-702
- https://doi.org/10.1016/s0003-4975(99)01524-6
Abstract
No abstract availableKeywords
This publication has 16 references indexed in Scilit:
- Endothelial dysfunction in cerebral microcirculation during hypothermic cardiopulmonary bypass in newborn lambsThe Journal of Thoracic and Cardiovascular Surgery, 1998
- Endothelial Cell Injury in Cardiovascular Surgery: The Systemic Inflammatory Response1The Annals of Thoracic Surgery, 1997
- Endothelial cell injury in cardiovascular surgery: The pathophysiology of vasomotor dysfunctionThe Annals of Thoracic Surgery, 1996
- Cardiopulmonary bypass, myocardial management, and support techniques Changes in autonomic response of the cerebral circulation after normothermic extracorporeal circulationThe Journal of Thoracic and Cardiovascular Surgery, 1996
- Moderate hypothermia reduces cardiopulmonary bypass-induced impairment of cerebrovascular responses to platelet productsThe Annals of Thoracic Surgery, 1996
- Effects of cardiopulmonary bypass and circulatory arrest on endothelium-dependent vasodilatation in the lungThe Journal of Thoracic and Cardiovascular Surgery, 1996
- Reperfusion injury induces apoptosis in rabbit cardiomyocytes.Journal of Clinical Investigation, 1994
- Induction of Endothelial Cell Apoptosis by TNFα: Modulation by Inhibitors of Protein SynthesisExperimental Cell Research, 1994
- Effect of deep hypothermia and circulatory arrest on cerebral blood flow and metabolismThe Annals of Thoracic Surgery, 1993
- Diminished basal nitric oxide release after myocardial ischemia and reperfusion promotes neutrophil adherence to coronary endothelium.Circulation Research, 1993