Inhibition of NFκB Induces Caspase-Independent Cell Death in Human T Lymphocytes
- 5 October 2001
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 287 (4), 895-899
- https://doi.org/10.1006/bbrc.2001.5678
Abstract
No abstract availableKeywords
This publication has 19 references indexed in Scilit:
- Inhibition of NF-κB Activity Enhances TRAIL Mediated Apoptosis in Breast Cancer Cell LinesBreast Cancer Research and Treatment, 2000
- Linkage of the BH4 Domain of Bcl-2 and the Nuclear Factor κB Signaling Pathway for Suppression of ApoptosisPublished by Elsevier ,1999
- The Crystal Structure of the IκBα/NF-κB Complex Reveals Mechanisms of NF-κB InactivationCell, 1998
- Structure of an IκBα/NF-κB ComplexCell, 1998
- NF-κB Antiapoptosis: Induction of TRAF1 and TRAF2 and c-IAP1 and c-IAP2 to Suppress Caspase-8 ActivationScience, 1998
- Mitochondria and ApoptosisScience, 1998
- The apoptosis-necrosis paradox. Apoptogenic proteases activated after mitochondrial permeability transition determine the mode of cell deathOncogene, 1997
- Sequential activation of ICE-like and CPP32-like proteases during Fas-mediated apoptosisNature, 1996
- Molecular Ordering of the Cell Death Pathway: Bcl-2 AND Bcl-xL FUNCTION UPSTREAM OF THE CED-3-LIKE APOPTOTIC PROTEASESPublished by Elsevier ,1996
- Reduction in mitochondrial potential constitutes an early irreversible step of programmed lymphocyte death in vivo.The Journal of Experimental Medicine, 1995