Smad-mediated Transcription Is Required for Transforming Growth Factor-β1-induced p57Kip2 Proteolysis in Osteoblastic Cells
Open Access
- 1 April 2001
- journal article
- Published by Elsevier
- Vol. 276 (14), 10700-10705
- https://doi.org/10.1074/jbc.m007499200
Abstract
No abstract availableKeywords
This publication has 32 references indexed in Scilit:
- Regulation of Smad ActivityCell, 2000
- SCF and Cullin/RING H2-Based Ubiquitin LigasesAnnual Review of Cell and Developmental Biology, 1999
- p57Kip2 Is Degraded through the Proteasome in Osteoblasts Stimulated to Proliferation by Transforming Growth Factor β1Journal of Biological Chemistry, 1999
- TGF-β SIGNAL TRANSDUCTIONAnnual Review of Biochemistry, 1998
- THE UBIQUITIN SYSTEMAnnual Review of Biochemistry, 1998
- The Role of p38 MAP Kinase in TGF-β1-Induced Signal Transduction in Human NeutrophilsBiochemical and Biophysical Research Communications, 1998
- Evidence for a Role of Rho-like GTPases and Stress-activated Protein Kinase/c-Jun N-terminal Kinase (SAPK/JNK) in Transforming Growth Factor β-mediated SignalingPublished by Elsevier ,1997
- Transforming Growth Factor β Activation of p44mapk in Proliferating Cultures of Epithelial CellsJournal of Biological Chemistry, 1995
- Cloning of a TGF$beta; type I receptor that forms a heteromeric complex with the TGF$beta; type II receptorCell, 1993
- Osteoblasts synthesize and respond to transforming growth factor-type beta (TGF-beta) in vitro.The Journal of cell biology, 1987