Hypersensitivity phenotypes associated with genetic and synthetic inhibitor-induced base excision repair deficiency
- 1 April 2007
- journal article
- review article
- Published by Elsevier in DNA Repair
- Vol. 6 (4), 530-543
- https://doi.org/10.1016/j.dnarep.2006.10.016
Abstract
No abstract availableKeywords
This publication has 96 references indexed in Scilit:
- Mapping of the Interaction Interface of DNA Polymerase β with XRCC1Structure, 2002
- Hypersensitivity of DNA polymerase β null mouse fibroblasts reflects accumulation of cytotoxic repair intermediates from site-specific alkyl DNA lesionsDNA Repair, 2002
- Direct Interaction between Mammalian DNA Polymerase β and Proliferating Cell Nuclear AntigenJournal of Biological Chemistry, 2002
- Combined effects of adenovirus-mediated wild-type p53 transduction, temozolomide and poly (ADP-ribose) polymerase inhibitor in mismatch repair deficient and non-proliferating tumor cellsCell Death & Differentiation, 2001
- A phase II study of temozolomide vs. procarbazine in patients with glioblastoma multiforme at first relapseBritish Journal of Cancer, 2000
- Role of a BRCT domain in the interaction of DNA ligase III-α with the DNA repair protein XRCC1Current Biology, 1998
- Post-translational modification of poly(ADP-ribose) polymerase induced by DNA strand breaksTrends in Biochemical Sciences, 1995
- Animal cell DNA polymerases in DNA repairMutation Research/DNA Repair, 1990
- Distribution of methyl and ethyl adducts following alkylation with monofunctional alkylating agentsMutation Research, 1990
- Methylation-induced blocks to in vitro DNA replicationMutation Research, 1985