Abstract
Some foreign compounds increase synthesis and excretion of ascorbic acid in rats. A new method for the preparation of L-galactono-gamma-lactone by high-pressure catalytic reduction of D-galacturonic acid is described. The rate of conversion of L-galactono-gamma-lactone into ascorbic acid was the same in microsomes from normal rats and rats treated with 1,2-5,6-dibenzanthracene, which caused increased ascorbic acid excretion in the whole animal.