Impact of α-bungarotoxin on transmitter release at the neuromuscular junction of the rat
- 13 October 1995
- journal article
- Published by Elsevier in Neuroscience Letters
- Vol. 199 (1), 49-52
- https://doi.org/10.1016/0304-3940(95)12013-t
Abstract
No abstract availableKeywords
This publication has 13 references indexed in Scilit:
- ATP released together with acetylcholine as the mediator of neuromuscular depression at frog motor nerve endings.The Journal of Physiology, 1994
- Adaptation of quantal content to decreased postsynaptic sensitivity at single endplates in alpha‐bungarotoxin‐treated rats.The Journal of Physiology, 1992
- Nicotinic receptors on the rat phrenic nerve: Evidence for negative feedbackNeuroscience Letters, 1991
- Run-down of neuromuscular transmission during repetitive nerve activity by nicotinic antagonists is not due to desensitization of the postsynaptic receptorBritish Journal of Pharmacology, 1991
- The nature of the presynaptic effects of (+)-tubocurarine at the mouse neuromuscular junction.The Journal of Physiology, 1988
- Feedback control of transmitter release at the neuromuscular junctionTrends in Pharmacological Sciences, 1988
- Dissociation of the end-plate potential run-down and the tetanic fade from the postsynaptic inhibition of acetylcholine receptor by α-neurotoxinsExperimental Neurology, 1987
- Presynaptic function modified by acetylcholine-receptor interaction in experimental autoimmune myasthenia gravisJournal of the Neurological Sciences, 1984
- Absence of [125I] alpha-bungarotoxin binding to motor nerve terminals of frog, lizard and mouse muscleJournal of Neuroscience, 1983
- α-Bungarotoxin enhances transmitter ‘released’ at the neuromuscular junctionNature, 1978