Guanosine 5′-O-(3-thiotriphosphate) causes endothelium-dependent, pertussis toxin-sensitive relaxations in porcine coronary arteries
- 30 November 1992
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 189 (1), 324-329
- https://doi.org/10.1016/0006-291x(92)91561-4
Abstract
No abstract availableKeywords
This publication has 16 references indexed in Scilit:
- Specific receptor-guanine nucleotide binding protein interaction mediates the release of endothelium-derived relaxing factor.Circulation Research, 1992
- Cholera toxin augments the release of endothelium-derived relaxing factor evoked by bradykinin and the calcium ionophore A23187General Pharmacology: The Vascular System, 1992
- Urea Gradient/SDS-Page; A Useful Tool in the Investigation of Signal Transducing G ProteinsJournal of Receptor Research, 1991
- Pertussis toxin inhibits endothelium-dependent relaxations evoked by fluorideEuropean Journal of Pharmacology, 1990
- Nω-nitro-L-arginine: a potent inhibitor of endothelium-derived relaxing factor formationEuropean Journal of Pharmacology, 1990
- Endothelial inositol phosphate generation and prostacyclin production in response to G-protein activation by AlF4−Biochemical Journal, 1989
- Guanosine 5‵-O-(3-thiotrisphosphate) potentiates both thrombin- and platelet-derived growth factor-induced inositol phosphate release in permeabilized vascular smooth muscle cellsPublished by Elsevier ,1989
- Pertussis toxin inhibits endothelium-dependent relaxations to certain agonists in porcine coronary arteries.The Journal of Physiology, 1989
- Vascular endothelial cells synthesize nitric oxide from L-arginineNature, 1988
- Activation of Turkey erythrocyte adenylate cyclase and blocking of the catecholamine-stimulated GTPase by guanosine 5′-(γ-thio) triphosphateBiochemical and Biophysical Research Communications, 1977