Huntingtin Acts in the Nucleus to Induce Apoptosis but Death Does Not Correlate with the Formation of Intranuclear Inclusions
- 1 October 1998
- Vol. 95 (1), 55-66
- https://doi.org/10.1016/s0092-8674(00)81782-1
Abstract
No abstract availableKeywords
This publication has 36 references indexed in Scilit:
- Aggregation of N-terminal huntingtin is dependent on the length of its glutamine repeatsHuman Molecular Genetics, 1998
- Intranuclear Neuronal Inclusions in Huntington's Disease and Dentatorubral and Pallidoluysian Atrophy: Correlation between the Density of Inclusions andIT15CAG Triplet Repeat LengthNeurobiology of Disease, 1998
- Aggregation of Huntingtin in Neuronal Intranuclear Inclusions and Dystrophic Neurites in BrainScience, 1997
- Exon 1 of the HD Gene with an Expanded CAG Repeat Is Sufficient to Cause a Progressive Neurological Phenotype in Transgenic MiceCell, 1996
- Role of the Ubiquitin-Proteasome Pathway in Regulating Abundance of the Cyclin-Dependent Kinase Inhibitor p27Science, 1995
- Widespread expression of Huntington's disease gene (IT15) protein productNeuron, 1995
- Cellular localization of the Huntington's disease protein and discrimination of the normal and mutated formNature Genetics, 1995
- Huntingtin is a cytoplasmic protein associated with vesicles in human and rat brain neuronsNeuron, 1995
- A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington's disease chromosomesCell, 1993
- Huntingtons DiseaseNew England Journal of Medicine, 1986