Vitamin D Arrests Thyroid Carcinoma Cell Growth and Induces p27 Dephosphorylation and Accumulation through PTEN/Akt-Dependent and -Independent Pathways
- 28 February 2002
- journal article
- Published by Elsevier in The American Journal of Pathology
- Vol. 160 (2), 511-519
- https://doi.org/10.1016/s0002-9440(10)64870-5
Abstract
No abstract availableKeywords
This publication has 45 references indexed in Scilit:
- PTEN expression is reduced in a subset of sporadic thyroid carcinomas: evidence that PTEN-growth suppressing activity in thyroid cancer cells is mediated by p27kip1Oncogene, 2000
- Overexpressed cyclin D3 contributes to retaining the growth inhibitor p27 in the cytoplasm of thyroid tumor cellsJournal of Clinical Investigation, 1999
- p27Kip1 ubiquitination and degradation is regulated by the SCFSkp2 complex through phosphorylated Thr187 in p27Current Biology, 1999
- Rapid actions of calcitriol and its side chain analogues CB1093 and GS1500 on intracellular calcium levels in skeletal muscle cells: a comparative studyBritish Journal of Pharmacology, 1999
- Mutation ofPten/Mmac1in mice causes neoplasia in multiple organ systemsProceedings of the National Academy of Sciences, 1999
- Effect of 22-Oxa-1,25-Dihydroxyvitamin D3 on Human Thyroid Cancer Cell Growth.Endocrine Journal, 1999
- Germline PTEN mutations in Cowden syndrome-like families.Journal of Medical Genetics, 1998
- High cancer susceptibility and embryonic lethality associated with mutation of the PTEN tumor suppressor gene in miceCurrent Biology, 1998
- Alterations of PTEN/MMAC1, a candidate tumor suppressor gene, and its homologue, PTH2, in small cell lung cancer cell linesOncogene, 1998
- Role of the Ubiquitin-Proteasome Pathway in Regulating Abundance of the Cyclin-Dependent Kinase Inhibitor p27Science, 1995