Obesity, Overeating, and Rapid Gastric Emptying in Rats with Ventromedial Hypothalamic Lesions
- 7 February 1986
- journal article
- research article
- Published by American Association for the Advancement of Science (AAAS) in Science
- Vol. 231 (4738), 609-611
- https://doi.org/10.1126/science.3511527
Abstract
Measurements confirm the quantitative theoretical prediction that the autonomic nonendocrine abnormality of rapid daytime gastric emptying is the major primary cause of the obesity resulting from ventromedial hypothalamic lesions in rats. Therapy for obesity could include slowing of stomach emptying.This publication has 41 references indexed in Scilit:
- Inhibition of gastric emptying by peripheral and central fenfluramine in rats: Correlation with anorexiaLife Sciences, 1984
- Apparent movement in the human visual fieldPhysiology & Behavior, 1983
- Gastrointestinal and metabolic consequences of a rat's meal on maintenance diet ad libitumPhysiology & Behavior, 1981
- Neurochemistry of appetite mechanismsProceedings Of The Nutrition Society, 1978
- Immediate effect of lesion of the ventromedial hypothalamic area upon glucose-induced insulin secretion in anaesthetized ratsDiabetologia, 1977
- Vagotomy in the rat reduces meal size of diets containing fatPhysiology & Behavior, 1974
- Hypothalamic Obesity: The Myth of the Ventromedial NucleusScience, 1973
- Postabsorptively induced suppression of appetite and the energostatic control of feedingPhysiology & Behavior, 1972
- Satiety and behavioral caloric compensation following intragastric glucose loads in the rat.Journal of Comparative and Physiological Psychology, 1972
- The VMH: A center for affective reactions, satiety, or both?Physiology & Behavior, 1965