Unaltered aconitase activity, but decreased complex I activity in substantia nigra pars compacta of patients with Parkinson's disease
- 19 March 1994
- journal article
- Published by Elsevier in Neuroscience Letters
- Vol. 169 (1-2), 126-128
- https://doi.org/10.1016/0304-3940(94)90372-7
Abstract
No abstract availableKeywords
This publication has 20 references indexed in Scilit:
- Iron‐Melanin Complex in Substantia Nigra of Parkinsonian Brains: An X‐Ray MicroanalysisJournal of Neurochemistry, 1992
- Oxidative stress as a cause of nigral cell death in Parkinson's disease and incidental lewy body diseaseAnnals of Neurology, 1992
- Normal Mitochondrial Genome in Brain from Patients with Parkinson's Disease and Complex I DefectJournal of Neurochemistry, 1990
- Mitochondrial Complex I Deficiency in Parkinson's DiseaseJournal of Neurochemistry, 1990
- Transition Metals, Ferritin, Glutathione, and Ascorbic Acid in Parkinsonian BrainsJournal of Neurochemistry, 1989
- Increased iron (III) and total iron content in post mortem substantia nigra of parkinsonian brainJournal of Neural Transmission, 1988
- INCREASED NIGRAL IRON CONTENT IN POSTMORTEM PARKINSONIAN BRAINThe Lancet, 1987
- Effects of 1‐Methyl‐4‐Phenyl‐1,2,3,6‐Tetrahydropyridine and 1‐Methyl‐4‐Phenylpyridinium Ion on Activities of the Enzymes in the Electron Transport System in Mouse BrainJournal of Neurochemistry, 1987
- Inhibition of NADH-linked oxidation in brain mitochondria by 1-methyl-4-phenyl-pyridine, a metabolite of the neurotoxin, 1-methyl-4-phenyl-1,2,5,6-tetrahydropyridineLife Sciences, 1985
- AN ELECTRON-TRANSPORT SYSTEM ASSOCIATED WITH THE OUTER MEMBRANE OF LIVER MITOCHONDRIAThe Journal of cell biology, 1967