Mitochondrial Autophagy Is an HIF-1-dependent Adaptive Metabolic Response to Hypoxia
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Open Access
- 1 April 2008
- journal article
- research article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 283 (16), 10892-10903
- https://doi.org/10.1074/jbc.m800102200
Abstract
No abstract availableThis publication has 45 references indexed in Scilit:
- BNIP3 Is an RB/E2F Target Gene Required for Hypoxia-Induced AutophagyMolecular and Cellular Biology, 2007
- Cardiac autophagy is a maladaptive response to hemodynamic stressJournal of Clinical Investigation, 2007
- Oxygen, a source of life and stressFEBS Letters, 2007
- Functional and physical interaction between Bcl-XL and a BH3-like domain in Beclin-1The EMBO Journal, 2007
- The transcription factor HIF-1α plays a critical role in the growth factor-dependent regulation of both aerobic and anaerobic glycolysisGenes & Development, 2007
- Multiple Factors Affecting Cellular Redox Status and Energy Metabolism Modulate Hypoxia-Inducible Factor Prolyl Hydroxylase Activity In Vivo and In VitroMolecular and Cellular Biology, 2007
- Disruption of the Arnt gene in endothelial cells causes hepatic vascular defects and partial embryonic lethality in miceHepatology, 2006
- Dioxygenases as O2-dependent regulators of the hypoxic response pathwayBiochemical and Biophysical Research Communications, 2005
- Signalling hypoxia by HIF hydroxylasesBiochemical and Biophysical Research Communications, 2005
- The von Hippel–Lindau protein, HIF hydroxylation, and oxygen sensingBiochemical and Biophysical Research Communications, 2005