Sodium requirement in secretory processes regulated through muscarinic receptors in rat parotid glands
- 1 June 1979
- journal article
- Published by Wiley in FEBS Letters
- Vol. 102 (1), 4-8
- https://doi.org/10.1016/0014-5793(79)80916-3
Abstract
No abstract availableThis publication has 11 references indexed in Scilit:
- Lanthanum as a Tool to Study the Role of Phosphatidylinositol in the Calcium Transport in Rat Parotid Glands upon Cholinergic StimulationEuropean Journal of Biochemistry, 1978
- Effects of ions on amylase release by dissociated pancreatic acinar cellsAmerican Journal of Physiology-Legacy Content, 1976
- Cholinergically stimulated phosphatidylinositol breakdown in parotid-gland fragments is independent of the ionic environmentBiochemical Journal, 1976
- The relationship of calcium to receptor-controlled stimulation of phosphatidylinositol turnover. Effects of acetylcholine, adrenaline, calcium ions, cinchocaine and a bivalent cation ionophore on rat parotid-gland fragmentsBiochemical Journal, 1975
- Breakdown of phosphatidylinositol provoked by muscarinic cholinergic stimulation of rat parotid-gland fragmentsBiochemical Journal, 1974
- The calcium ionophore a 23 187 as a probe for studying the role of Ca2+ ions in the mediation of carbachol effects on rat salivary glands: protein secretion and metabolism of phospholipids and glycogenFEBS Letters, 1974
- A Calcium Ionophore Simulating the Action of Epinephrine on the α-Adrenergic ReceptorProceedings of the National Academy of Sciences, 1974
- Potassium Ion Release and Enzyme Secretion: Adrenergic Regulation by α-and β-ReceptorsScience, 1971
- Energy- and sodium-dependent uptake of inositol by kidney cortex slicesBiochemical and Biophysical Research Communications, 1965
- [17] Amylases, α and βMethods in Enzymology, 1955