Exercise in the Disposition of Dietary Calories

Abstract

THE evidence relating the level of the serum lipids to the etiology of human atherosclerosis is indirect.¹ The association of atherosclerosis or its clinical consequences with a record of persistent hypercholesteremia has been observed on many occasions and in both natural and experimental conditions. The relation exists in several species although the degree of association appears to be species specific.^{2 , 3} The development of refined biophysical methods for the characterization of serum lipids has not greatly clarified this relation of serum lipids to disease although the new methods have emphasized a lack of correlation between the serum cholesterol and lipoprotein quantities . . .