The role of the nervous system in the production and maintenance of blood pressure has long been recognized. It has always been difficult, however, to relate experimental hypertension produced by acute and chronic experiments on the nervous system to the clinical disorders in which elevation of blood pressure occurs. Recently, renewed interest in the role of the central nervous system has been manifested (1, 2), and several excellent reviews of the present status of the physiological evidence relating the central nervous system to the cardiovascular system have become available (3-5). The increasing ability to separate out certain specific causes of