Dual MAP kinase pathways mediate opposing forms of long-term plasticity at CA3–CA1 synapses
- 1 November 2000
- journal article
- research article
- Published by Springer Nature in Nature Neuroscience
- Vol. 3 (11), 1107-1112
- https://doi.org/10.1038/80624
Abstract
No abstract availableKeywords
This publication has 39 references indexed in Scilit:
- ERK Plays a Regulatory Role in Induction of LTP by Theta Frequency Stimulation and Its Modulation by β-Adrenergic ReceptorsNeuron, 1999
- Neuronal Activity-Dependent Cell Survival Mediated by Transcription Factor MEF2Science, 1999
- P/Q-type calcium channels mediate the activity-dependent feedback of syntaxin-1ANature, 1999
- MAP kinase pathwaysProgress in Biophysics and Molecular Biology, 1999
- Rap1 mediates sustained MAP kinase activation induced by nerve growth factorNature, 1998
- Activation of p42 Mitogen-activated Protein Kinase in Hippocampal Long Term PotentiationJournal of Biological Chemistry, 1996
- Opposing Effects of ERK and JNK-p38 MAP Kinases on ApoptosisScience, 1995
- Involvement of a calcineurin/ inhibitor-1 phosphatase cascade in hippocampal long-term depressionNature, 1994
- Mechanisms underlying induction of homosynaptic long-term depression in area CA1 of the hippocampusNeuron, 1992
- Homosynaptic long-term depression in area CA1 of hippocampus and effects of N-methyl-D-aspartate receptor blockade.Proceedings of the National Academy of Sciences, 1992