Mutations in Exon 11 of c-Kit Occur Preferentially in Malignant versus Benign Gastrointestinal Stromal Tumors and Do Not Occur in Leiomyomas or Leiomyosarcomas
- 1 January 1999
- journal article
- research article
- Published by Elsevier in The American Journal of Pathology
- Vol. 154 (1), 53-60
- https://doi.org/10.1016/s0002-9440(10)65250-9
Abstract
No abstract availableKeywords
This publication has 25 references indexed in Scilit:
- Gain-of-Function Mutations of c- kit in Human Gastrointestinal Stromal TumorsScience, 1998
- The protooncogene c-kit and c-kit ligand in human disease☆☆☆★Journal of Allergy and Clinical Immunology, 1997
- Sequence Analysis of Two Genomic Regions Containing the KIT and the FMS Receptor Tyrosine Kinase GenesGenomics, 1997
- A case for interstitial cells of Cajal as pacemakers and mediators of neurotransmission in the gastrointestinal tractGastroenterology, 1996
- Gastrointestinal Stromal Tumors—Value of CD34 Antigen in their Identification and Separation from True Leiomyomas and SchwannomasThe American Journal of Surgical Pathology, 1995
- CD34 immunoexpression in stromal tumours of the gastrointestinal tract and in mesenteric fibromatosesHistopathology, 1994
- Preferential localization of c-kit product in tissue mast cells, basal cells of skin, epithelial cells of breast, small cell lung carcinoma and seminoma/dysgerminoma in human: immunohistochemical study on formalin-fixed, paraffin-embedded tissuesVirchows Archiv, 1994
- Identification of mutations in the coding sequence of the proto-oncogene c-kit in a human mast cell leukemia cell line causing ligand-independent activation of c-kit product.Journal of Clinical Investigation, 1993
- Stem cell factor is encoded at the SI locus of the mouse and is the ligand for the c-kit tyrosine kinase receptorCell, 1990
- Identification of a ligand for the c-kit proto-oncogeneCell, 1990