A Protein Interaction Network Links GIT1, an Enhancer of Huntingtin Aggregation, to Huntington's Disease
- 1 September 2004
- journal article
- Published by Elsevier in Molecular Cell
- Vol. 15 (6), 853-865
- https://doi.org/10.1016/j.molcel.2004.09.016
Abstract
No abstract availableKeywords
This publication has 38 references indexed in Scilit:
- ADP-Ribosylation Factor 6 and a Functional PIX/p95-APP1 Complex Are Required for Rac1B-mediated Neurite OutgrowthMolecular Biology of the Cell, 2003
- Analyzing yeast protein–protein interaction data obtained from different sourcesNature Biotechnology, 2002
- Cell migration: GAPs between membrane traffic and the cytoskeletonEMBO Reports, 2001
- Loss of normal huntingtin function: new developments in Huntington's disease researchTrends in Neurosciences, 2001
- p95-APP1 links membrane transport to Rac-mediated reorganization of actinNature Cell Biology, 2000
- Multiple endocytic pathways of G protein-coupled receptors delineated by GIT1 sensitivityProceedings of the National Academy of Sciences, 2000
- The Cell as a Collection of Protein Machines: Preparing the Next Generation of Molecular BiologistsCell, 1998
- Intranuclear Neuronal Inclusions in Huntington's Disease and Dentatorubral and Pallidoluysian Atrophy: Correlation between the Density of Inclusions andIT15CAG Triplet Repeat LengthNeurobiology of Disease, 1998
- Aggregation of Huntingtin in Neuronal Intranuclear Inclusions and Dystrophic Neurites in BrainScience, 1997
- Formation of Neuronal Intranuclear Inclusions Underlies the Neurological Dysfunction in Mice Transgenic for the HD MutationCell, 1997