Nuclear Factor-κB1 (p50) Limits the Inflammatory and Fibrogenic Responses to Chronic Injury
- 1 March 2005
- journal article
- Published by Elsevier in The American Journal of Pathology
- Vol. 166 (3), 695-708
- https://doi.org/10.1016/s0002-9440(10)62291-2
Abstract
No abstract availableKeywords
This publication has 38 references indexed in Scilit:
- Shaping the nuclear action of NF-κBNature Reviews Molecular Cell Biology, 2004
- RelB Forms Transcriptionally Inactive Complexes with RelA/p65Journal of Biological Chemistry, 2003
- Gene deletion of NF-κB p50 does not alter the hepatic inflammatory response to ischemia/reperfusionJournal of Hepatology, 2002
- CD40 Induces Interleukin-6 Gene Transcription in Dendritic CellsPublished by Elsevier ,2002
- Interactions between Hepatic Stellate Cells and the Immune SystemSeminars in Liver Disease, 2001
- Activators and target genes of Rel/NF-κB transcription factorsOncogene, 1999
- Inhibition of NFκB in activated rat hepatic stellate cells by proteasome inhibitors and an IκB super-repressorHepatology, 1998
- NF-κB AND REL PROTEINS: Evolutionarily Conserved Mediators of Immune ResponsesAnnual Review of Immunology, 1998
- p50–NF-κB Complexes Partially Compensate for the Absence of RelB: Severely Increased Pathology in p50−/−relB−/−Double-knockout MiceThe Journal of Experimental Medicine, 1997
- Multiorgan inflammation and hematopoietic abnormalities in mice with a targeted disruption of RelB, a member of the NF-κB/Rel familyCell, 1995