A pyridazinyl derivative of gamma-aminobutyric acid (GABA), SR 95531, is a potent antagonist of Cl− channel opening regulated by GABAA receptors
- 1 July 1987
- journal article
- research article
- Published by Elsevier in Neuropharmacology
- Vol. 26 (7), 779-783
- https://doi.org/10.1016/0028-3908(87)90242-5
Abstract
No abstract availableKeywords
This publication has 8 references indexed in Scilit:
- Modulation of gamma-aminobutyric acid-mediated inhibitory synaptic currents in dissociated cortical cell cultures.Proceedings of the National Academy of Sciences, 1986
- Specific binding of a phenyl-pyridazinium derivative endowed with GABAA receptor antagonist activity to rat brainNeuropharmacology, 1986
- Interactions between three pyridazinyl-GABA derivatives and central GABA and glycine receptors in the rat, an in vivo microiontophoretic studyNeuropharmacology, 1986
- Biochemical characterization of the interaction of three pyridazinyl-GABA derivatives with the GABAA receptor siteBrain Research, 1986
- Pyridazinyl-GABA derivatives as GABA and glycine antagonists in the spinal cord of the catNeuroscience Letters, 1986
- Ionic mechanism of muscarinic cholinergic depolarization of mouse spinal cord neurons in cell cultureJournal of Neurophysiology, 1983
- Improved patch-clamp techniques for high-resolution current recording from cells and cell-free membrane patchesPflügers Archiv - European Journal of Physiology, 1981