The Cytoprotective Effects of the Glycoprotein 130 Receptor-coupled Cytokine, Cardiotrophin-1, Require Activation of NF-κB
Open Access
- 1 October 2001
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 276 (40), 37621-37629
- https://doi.org/10.1074/jbc.m103276200
Abstract
No abstract availableKeywords
This publication has 69 references indexed in Scilit:
- Akt Stimulates the Transactivation Potential of the RelA/p65 Subunit of NF-κB through Utilization of the IκB Kinase and Activation of the Mitogen-activated Protein Kinase p38Journal of Biological Chemistry, 2001
- The Heart Is a Source of Circulating Cardiotrophin-1 in HumansBiochemical and Biophysical Research Communications, 2000
- Involvement of a p38 Mitogen-activated Protein Kinase Phosphatase in Protecting Neonatal Rat Cardiac Myocytes from IschemiaJournal of Molecular and Cellular Cardiology, 2000
- Hypoxic Stress Induces Cardiotrophin-1 Expression in Cardiac MyocytesBiochemical and Biophysical Research Communications, 1999
- Induction of NF-κB by the Akt/PKB kinaseCurrent Biology, 1999
- p38 and Extracellular Signal-regulated Kinase Mitogen-activated Protein Kinase Pathways Are Required for Nuclear Factor-κB p65 Transactivation Mediated by Tumor Necrosis FactorJournal of Biological Chemistry, 1998
- Activation of Nuclear Transcription Factor NF-κB by Interleukin-1 Is Accompanied by Casein Kinase II-mediated Phosphorylation of the p65 SubunitJournal of Biological Chemistry, 1997
- Intracellular signalling: Putting JAKs on the kinase MAPCurrent Biology, 1996
- Opposing Effects of ERK and JNK-p38 MAP Kinases on ApoptosisScience, 1995
- Pyridinyl imidazoles inhibit IL-1 and TNF production at the protein levelInflammation Research, 1993