Susceptibility of spontaneous sympathetic outflow and sympathetic reflexes to depression by clonidine
- 1 July 1977
- journal article
- research article
- Published by Elsevier in European Journal of Pharmacology
- Vol. 44 (2), 121-129
- https://doi.org/10.1016/0014-2999(77)90098-x
Abstract
No abstract availableThis publication has 29 references indexed in Scilit:
- Cardiovascular and electrophysiological responses to sympathetic pathway stimulationAmerican Journal of Physiology-Legacy Content, 1976
- Conduction in descending spinal pathways initiated by somatosympathetic reflexesAmerican Journal of Physiology-Legacy Content, 1975
- Influence of heart rate on the reduction of sympathetic outflow produced by L-dopaAmerican Journal of Physiology-Legacy Content, 1974
- Modification by antihypertensive drugs of reflex circulatory responses induced by vertical tilting or bilateral carotid occlusionEuropean Journal of Pharmacology, 1973
- Effects of clonidine on aortic diameter and aortic baroreceptor activityEuropean Journal of Pharmacology, 1972
- Evoked splanchnic potentials produced by electrical stimulation of medullary vasomotor regionsExperimental Brain Research, 1971
- Evidence for a central noradrenaline receptor stimulation by clonidineLife Sciences, 1970
- Studies of the new hypotensive drug ST 155European Journal of Pharmacology, 1968
- Analysis of the cardiovascular effects of 2-(2,6-dichlorophenylamino)-2-imidazoline hydrochloride (Catapres)European Journal of Pharmacology, 1968
- Cardiovascular effects of 2-(2,6-dichlorophenylamino)-2-imidazoline hydrochloride (ST 155)European Journal of Pharmacology, 1968