Contribution of the IsK (MinK) Potassium Channel Subunit to Regulatory Volume Decrease in Murine Tracheal Epithelial Cells
Open Access
- 1 November 2000
- journal article
- Published by Elsevier
- Vol. 275 (45), 34849-34852
- https://doi.org/10.1074/jbc.c000633200
Abstract
No abstract availableKeywords
This publication has 32 references indexed in Scilit:
- Characterisation of a cell swelling‐activated K+‐selective conductance of Ehrlich mouse ascites tumour cellsThe Journal of Physiology, 2000
- Osmosensitive Cl− currents and their relevance to regulatory volume decrease in human intestinal T84 cells: outwardly vs. inwardly rectifying currentsThe Journal of Physiology, 1998
- Importance of basolateral K+ conductance in maintaining Cl− secretion in murine nasal and colonic epitheliaThe Journal of Physiology, 1998
- Calcium-activated potassium channelsCurrent Opinion in Neurobiology, 1998
- Role of the ISK protein in the IminK channel complexTrends in Pharmacological Sciences, 1997
- Positional cloning of a novel potassium channel gene: KVLQT1 mutations cause cardiac arrhythmiasNature Genetics, 1996
- Selective response of human airway epithelia to luminal but not serosal solution hypertonicity. Possible role for proximal airway epithelia as an osmolality transducer.Journal of Clinical Investigation, 1994
- Hypotonic solution increases the slowly activating potassium current IsK expressed in Xenopus oocytesBiochemical and Biophysical Research Communications, 1992
- Toxins in the characterization of potassium channelsTrends in Neurosciences, 1989
- Voltage-gated chloride currents in cultured canine tracheal epithelial cellsThe Journal of Membrane Biology, 1989