Epicutaneous Antigen Exposure Primes for Experimental Eosinophilic Esophagitis in Mice
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- 1 September 2005
- journal article
- Published by Elsevier in Gastroenterology
- Vol. 129 (3), 985-994
- https://doi.org/10.1053/j.gastro.2005.06.027
Abstract
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This publication has 53 references indexed in Scilit:
- Anti–interleukin-5 (mepolizumab) therapy for hypereosinophilic syndromes☆Journal of Allergy and Clinical Immunology, 2004
- Eosinophilic gastrointestinal disorders (EGID)☆Journal of Allergy and Clinical Immunology, 2004
- Intratracheal IL-13 induces eosinophilic esophagitis by an IL-5, eotaxin-1, and STAT6-dependent mechanism1Gastroenterology, 2003
- Stat6-Deficient Mice Develop Airway Hyperresponsiveness and Peribronchial Fibrosis during Chronic Fungal AsthmaThe American Journal of Pathology, 2002
- IL-13 induces eosinophil recruitment into the lung by an IL-5– and eotaxin-dependent mechanismJournal of Allergy and Clinical Immunology, 2001
- Abrogation of lung inflammation in sensitized Stat6‐deficient mice is dependent on the allergen inhalation procedureBritish Journal of Pharmacology, 2000
- Th2 responses induced by epicutaneous or inhalational protein exposure are differentially dependent on IL-4Journal of Clinical Investigation, 2000
- Essential role of Stat6 in IL-4 signallingNature, 1996
- The role of the eosinophil in gastrointestinal diseasesCurrent Opinion in Gastroenterology, 1995
- Atopic dermatitis: The skin as a window into the pathogenesis of chronic allergic diseasesJournal of Allergy and Clinical Immunology, 1995