STAT-1 Interacts with p53 to Enhance DNA Damage-induced Apoptosis
Open Access
- 1 February 2004
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 279 (7), 5811-5820
- https://doi.org/10.1074/jbc.m302637200
Abstract
No abstract availableKeywords
This publication has 39 references indexed in Scilit:
- The C-terminal activation domain of the STAT-1 transcription factor is necessary and sufficient for stress-induced apoptosisCell Death & Differentiation, 2002
- The Brn-3a transcription factor inhibits the pro-apoptotic effect of p53 and enhances cell cycle arrest by differentially regulating the activity of the p53 target genes encoding Bax and p21CIP1/Waf1Oncogene, 2002
- The C‐terminal activation domain of the STAT‐1 transcription enhances ischemia/reperfusion‐induced apoptosis in cardiac myocytesThe FASEB Journal, 2002
- Live or let die: the cell's response to p53Nature Reviews Cancer, 2002
- Stat1-Dependent, p53-Independent Expression of p21waf1 Modulates Oxysterol-Induced ApoptosisMolecular and Cellular Biology, 2002
- Ischemia-induced STAT-1 Expression and Activation Play a Critical Role in Cardiomyocyte ApoptosisJournal of Biological Chemistry, 2000
- Mdm2 promotes the rapid degradation of p53Nature, 1997
- Targeted Disruption of the Mouse Stat1 Gene Results in Compromised Innate Immunity to Viral DiseaseCell, 1996
- p53 Mutations in Human CancersScience, 1991
- Interferon ReviewInvestigative Radiology, 1987