Indole‐N‐Methylation of β‐Carbolines: The Brain's Bioactivation Route to Toxins in Parkinson's Disease?a,b
- 1 May 1992
- journal article
- Published by Wiley in Annals of the New York Academy of Sciences
- Vol. 648 (1), 263-265
- https://doi.org/10.1111/j.1749-6632.1992.tb24551.x
Abstract
No abstract availableThis publication has 7 references indexed in Scilit:
- Mitochondrial respiratory inhibition by N-methylated beta-carboline derivatives structurally resembling N-methyl-4-phenylpyridine.Proceedings of the National Academy of Sciences, 1990
- Biochemical mechanism of action of the dopaminergic neurotoxin 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP)Toxicology Letters, 1989
- A N-methyltransferase in human brain catalyses N-methylation of 1,2,3,4-tetrahydroisoquinoline into N-methyl-1,2,3,4-tetrahydroisoquinoline, a precursor of a dopaminergic neurotoxin, N-methylisoquinolinium ionBiochemical and Biophysical Research Communications, 1989
- In vivo dopaminergic neurotoxicity of the 2-β-methylcarbolinium ion, a potential endogenous MPP+ analogEuropean Journal of Pharmacology, 1988
- Inhibition of mitochondrial respiration by analogs of 4-phenylpyridine and 1-methyl-4-phenylpyridinium cation (MPP+), the neurotoxic metabolite of MPTPBiochemical and Biophysical Research Communications, 1987
- Reevaluation of the products of tryptamine catalyzed by rabbit liver N-methyltransferasesBiochemical Pharmacology, 1986
- β-Carboline analogues of N-methyl-4-phenyl-1,2,5,6-tetrahydropyridine (MPTP): Endogenous factors underlying idiopathic parkinsonism?Neuroscience Letters, 1985