Cdc2 and Cdk2 Kinase Activated by Transforming Growth Factor-β1 Trigger Apoptosis through the Phosphorylation of Retinoblastoma Protein in FaO Hepatoma Cells
Open Access
- 1 November 1999
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 274 (45), 31775-31783
- https://doi.org/10.1074/jbc.274.45.31775
Abstract
No abstract availableKeywords
This publication has 62 references indexed in Scilit:
- CDK4, a Possible Critical Regulator of Apoptosis in Rat Pheochromocytoma PC12 CellsBiochemical and Biophysical Research Communications, 1998
- ICE-like protease (caspase) is involved in transforming growth factor β1-mediated apoptosis in FaO rat hepatoma cell lineHepatology, 1998
- The E1B 19K protein associates with lamins in vivo and its proper localization is required for inhibition of apoptosisOncogene, 1997
- Differential regulation of p34cdc2 and p33cdk2 by transforming growth factor-β1 in murine mammary epithelial cellsJournal of Cellular Biochemistry, 1995
- p34 cdc2 and ApoptosisScience, 1995
- Chromatin condensation during apoptosis is accompanied by degradation of lamin A+B, without enhanced activation of cdc2 kinase.The Journal of cell biology, 1994
- Premature p34 cdc2 Activation Required for ApoptosisScience, 1994
- Negative Regulation of G1 in Mammalian Cells: Inhibition of Cyclin E-Dependent Kinase by TGF-βScience, 1993
- Requirement for a functional Rb-1 gene in murine developmentNature, 1992
- Induction of apoptosis in fibroblasts by c-myc proteinCell, 1992