Variations in the C3, C3a receptor, and C5 genes affect susceptibility to bronchial asthma
- 27 July 2004
- journal article
- research article
- Published by Springer Nature in Human Genetics
- Vol. 115 (4), 295-301
- https://doi.org/10.1007/s00439-004-1157-z
Abstract
No abstract availableKeywords
This publication has 29 references indexed in Scilit:
- A Polymorphism in the Human UGRP1 Gene Promoter That Regulates Transcription Is Associated with an Increased Risk of AsthmaAmerican Journal of Human Genetics, 2002
- Complement Factors C3a and C5a Are Increased in Bronchoalveolar Lavage Fluid after Segmental Allergen Provocation in Subjects with AsthmaAmerican Journal of Respiratory and Critical Care Medicine, 2001
- Genetics of allergen-induced asthmaJournal of Allergy and Clinical Immunology, 2001
- Expression of the Complement Anaphylatoxin C3a and C5a Receptors on Bronchial Epithelial and Smooth Muscle Cells in Models of Sepsis and AsthmaThe Journal of Immunology, 2001
- Mutation detection by TaqMan-allele specific amplification: Application to molecular diagnosis of glycogen storage disease type Ia and medium-chain acyl-CoA dehydrogenase deficiencyHuman Mutation, 2000
- A genome-wide search for asthma susceptibility loci in ethnically diverse populationsNature Genetics, 1997
- Interleukin 12 inhibits antigen-induced airway hyperresponsiveness, inflammation, and Th2 cytokine expression in mice.The Journal of Experimental Medicine, 1995
- Complement Peptides C3a- and C5a-Induced Mediator Release from Dissociated Human Skin Mast CellsJournal of Investigative Dermatology, 1994
- Guidelines for the diagnosis and management of asthma. National Heart, Lung, and Blood Institute. National Asthma Education Program. Expert Panel Report.1991
- THE VALIDITY OF APPROXIMATION METHODS FOR INTERVAL ESTIMATION OF THE ODDS RATIOAmerican Journal of Epidemiology, 1981